Marketing causality: identifying the path that manufactures false performance.

SUPPORTED DECISIONAuthorise a recommendation only if the main confounding paths are blocked, measured or explicitly left in uncertainty.

Directly verifiable evidence

Primary sources to check the key claims.

  1. Holland (1986), Statistics and Causal Inference (opens in a new tab)

    Definition of the causal problem and the counterfactual.

Evidence status

What was not reproduced.

The source's training was re-run and reproduces its predictive performance (holdout R² of 0.843). The graph of relationships between channels does not come out of it: it comes back undifferentiated, whereas the paper presents one whose links would suggest plausible causal pathways. This dossier therefore validates no causal conclusion, and that measurement supports with a figure what the page states: predicting well is not having established a structure.

    Key distinctions

    Three conditions before calculating.

    01

    An explicit estimand

    Average effect, effect on the treated, effect of a budget tranche or long-run effect are not the same quantity. The design cannot be chosen before the population, the action and the horizon have been defined.

    02

    Open paths

    A variable common to the action and to the outcome creates a non-causal path. Controlling for it can help, but controlling for a mediator or a collider can also introduce a new bias.

    03

    Design before model

    Randomisation, an eligibility rule, or credible geographic or temporal variation create the comparison. A more complex model does not automatically repair a missing counterfactual.

    Method

    Building a causal claim proportionate to the evidence

    1. 01Define the intervention

      State who receives what, when, at what dose and relative to which absence of action; a composite campaign must be decomposed if its elements can vary.

    2. 02Draw the causes

      Map prior variables, targeting criteria, latent demand, competition, mediators and outcome; the graph serves to decide what to measure and what not to control.

    3. 03Choose the variation

      Prefer randomisation; failing that, document the assumption that makes a variation credibly quasi-experimental and look for placebo tests or pre-trends.

    4. 04Bound the use

      Report the interval, the sensitivity to confounders and the population covered; local evidence must not be turned into a universal return.

    ORIGINAL ASSET

    The map of bias paths

    The graph separates the effect sought, reverse causality, selection and common variables. Each path calls for a different design or diagnostic.

    COMMON CAUSEForecast demand+€6 bias
    ↘
    ↙
    ACTIONMedia spend
    → effect sought →
    OUTCOMESales
    OMITTED VARIABLEPromotion + season+€5 bias
    ↗
    ↖
    ASSOCIATION€18
    −
    DOCUMENTED BIAS€11
    =
    TESTED EFFECT€7
    CSVDownload the causal-bias register↓
    ILLUSTRATIVE EXAMPLE · SIMULATED DATA

    An apparent return of €18 falls to €7 after identification

    01 · SITUATION

    A historical regression attributes €18 of sales to every thousand euros of media. Yet budgets are increased before high-demand weeks and during promotions.

    02 · CALCULATION

    The decomposition estimates €6 of bias linked to forecast demand, €3 to promotions and €2 to season, that is €11. A comparable geographic test measures an effect of €7, consistent with 18 − 11.

    03 · DECISION

    The usable causal effect is €7 within the scope of the test. The €18 return remains a descriptive association and must not guide the allocation.

    Acceptance conditions

    What must be true to act.

    1. 01

      Population, intervention, comparison, outcome and horizon are defined.

    2. 02

      The common causes of the action and the outcome are mapped.

    3. 03

      The design creates independent variation or defends a testable assumption.

    4. 04

      The final claim stays limited to the population and the range observed.

    Limits

    What this analysis does not prove.

    • A causal graph formalises assumptions; it does not make them true.
    • Effects can vary by segment, dose, period and interaction with other levers.
    • Sensitivity analyses reduce overconfidence but do not reveal an unmeasured confounder.

    References

    Works cited.

    1. Pearl, J. (2009), Causality: Models, Reasoning, and Inference.
    2. Hernán, M. A. & Robins, J. M. (2020), Causal Inference: What If.
    3. Shadish, W. R., Cook, T. D. & Campbell, D. T. (2002), Experimental and Quasi-Experimental Designs.